RS Belgrade Lalic NM

RS Belgrade Lalic NM

O2k-Network Lab	Clinic for Endocrinology, Diabetes and Metabolic Diseases Clinical Center of Serbia
Address	Faculty of Medicine University of Belgrade Dr Subotica 13, Belgrade, 11000
City	Belgrade
State/Prov
Country	Republic of Serbia
Weblink
Contact	Lalic Nebojsa M
Team	Krako Jakovljevic Nina, Krakov N, Pavlovic Kasja
Team previous
Status	O2k 2016 –
Oroboros Events	MiPNet15.12 IOC57 Druskininkai LT
Topics

O2k-Publications

	Published	Reference
Pesta 2021 Front Endocrinol (Lausanne)	2021	Pesta D, Jelenik T, Zaharia OP, Bobrov P, Görgens S, Bódis K, Karusheva Y, Krako Jakovljevic N, Lalic NM, Markgraf DF, Burkart V, Müssig K, Knebel B, Kotzka J, Eckel J, Strassburger K, Szendroedi J, Roden M (2021) NDUFB6 polymorphism is associated with physical activity-mediated metabolic changes in type 2 diabetes. https://doi.org/10.3389/fendo.2021.693683
Krako Jakovljevic 2021 Int J Mol Sci	2021	Krako Jakovljevic N, Pavlovic K, Jotic A, Lalic K, Stoiljkovic M, Lukic L, Milicic T, Macesic M, Stanarcic Gajovic J, Lalic NM (2021) Targeting mitochondria in diabetes. Int J Mol Sci 22:6642.

	Published	Reference
Calabria 2019 MiPschool Coimbra	2019	Investigating mitochondrial function in blood cells by high-resolution respirometry: contribution to an interlaboratory study. MitoEAGLE WG4.
Pavlovic 2019 MiP2019	2019	Effects of therapeutic metformin concentration on mitochondria in muscle cells.
Zujovic 2019 MiP2019	2019	Cell membrane permeabilization: digitonin optimization pitfalls.
Krako Jakovljevic 2019 MiP2019	2019	Lower insulin sensitivity differently affects mitochondrial coupling in liver and muscle cells.
Lalic 2109 MiP2019	2019	Pathophysiology of type 2 diabetes and mitochondrial dysfunction: possible therapeutic implications.
Pavlovic 2017 MITOEAGLE Obergurgl	2017	Establishing cellular model for studying mechanisms of muscle insulin resistance: towards understanding the role of mitochondria.
Krako 2016 Abstract MitoFit Science Camp 2016	2016	Conformational targeting of amyloid β oligomers inside endoplasmic reticulum rescues mitochondrial deficits in 7PA2 cells.