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|keyword=Dr. Morris investigates how the susceptibility to diet-induced weight gain is mediated by systemic and peripheral energy metabolism through the modulation of the neural centers regulating energy intake and expenditure. His work centers on how the function of the primary energy producing cellular bodies, mitochondria, can impact tissue function and ultimately systemic health. Currently, the lab is investigating: 1) how liver mitochondrial function can, through neural pathways to the brain, influence high fat diet-induced weight, 2) how systemic energy expenditure serves as an independent regulator of weight gain and adiposity, and 3) whether transcriptional control of mitochondria in the ventromedial hypothalamus is necessary for susceptibility to weight gain.
|keyword=Dr. Morris investigates how the susceptibility to diet-induced weight gain is mediated by systemic and peripheral energy metabolism through the modulation of the neural centers regulating energy intake and expenditure. His work centers on how the function of the primary energy producing cellular bodies, mitochondria, can impact tissue function and ultimately systemic health. Currently, the lab is investigating: 1) how liver mitochondrial function can, through neural pathways to the brain, influence high fat diet-induced weight, 2) how systemic energy expenditure serves as an independent regulator of weight gain and adiposity, and 3) whether transcriptional control of mitochondria in the ventromedial hypothalamus is necessary for susceptibility to weight gain.
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[[Category:Power-O2k]]

Latest revision as of 16:22, 3 November 2022

                



US KS Kansas City Morris EM

Oroboros O2k-Network

O2k-Network
O2k-Network Lab Department of Molecular & Integrative Physiology, University of Kansas Medical Center
Address Mail Stop 3043, 66160
City Kansas City
State/Prov Kansas (KS)
Country USA
Weblink https://www.kumc.edu/emorris2.html
Contact Morris E Matthew
Team
Team previous
Status 2 Power-O2k 2020-
Oroboros Events
Topics Dr. Morris investigates how the susceptibility to diet-induced weight gain is mediated by systemic and peripheral energy metabolism through the modulation of the neural centers regulating energy intake and expenditure. His work centers on how the function of the primary energy producing cellular bodies, mitochondria, can impact tissue function and ultimately systemic health. Currently, the lab is investigating: 1) how liver mitochondrial function can, through neural pathways to the brain, influence high fat diet-induced weight, 2) how systemic energy expenditure serves as an independent regulator of weight gain and adiposity, and 3) whether transcriptional control of mitochondria in the ventromedial hypothalamus is necessary for susceptibility to weight gain.


O2k-Publications

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O2k-Abstracts

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