Difference between revisions of "Template:Current week ecosystem agenda"
From Bioblast
| Line 5: | Line 5: | ||
:{| | :{| | ||
| width="110" align="center" | [[Image:O2k-Support.jpg|60px|link=http://wiki.oroboros.at/index.php/O2k-Open_Support |O2k-Open Support]][[File:Red-loss of data.png|25px|Warning: loss of data]] | | width="110" align="center" | [[Image:O2k-Support.jpg|60px|link=http://wiki.oroboros.at/index.php/O2k-Open_Support |O2k-Open Support]][[File:Red-loss of data.png|25px|Warning: loss of data]] | ||
|'''Can NADH be | |'''Can NADH be used as an externally added substrate to obtain the [[NADH electron transfer-pathway state]] in [[Mitochondrial_respiration |mitochondrial respiration]]? '''<br> | ||
No, | No, in most types of intact mitochondria NADH cannot pass the [[mitochondrial inner membrane]] (mtIM). Yeast mitochondria are an exception. <br> | ||
- »[[Nicotinamide_adenine_dinucleotide|Nicotinamide adenine dinucleotide]]« and »[[MitoPedia:_Substrates_and_metabolites|Substrates and metabolites]]« | - »[[Nicotinamide_adenine_dinucleotide|Nicotinamide adenine dinucleotide]]« and »[[MitoPedia:_Substrates_and_metabolites|Substrates and metabolites]]« | ||
|} | |} | ||
| Line 15: | Line 15: | ||
|'''Low-intensity exercise increases fat oxidation in mitochondria of blood mononuclear cells from sedentary adults'''<br> | |'''Low-intensity exercise increases fat oxidation in mitochondria of blood mononuclear cells from sedentary adults'''<br> | ||
:from the O2k-Network »[[LV Riga Makrecka-Kuka M]]« | :from the O2k-Network »[[LV Riga Makrecka-Kuka M]]« | ||
*Liepinsh E, Makarova E, Plakane L, Konrade I, Liepins K, Videja M, Sevostjanovs E, Grinberga S, Makrecka-Kuka M, Dambrova M (2020) Low-intensity exercise stimulates bioenergetics and increases fat oxidation in mitochondria of blood mononuclear cells from sedentary adults. Physiol Rep 8:e14489. [[Liepinsh 2020 Physiol Rep|»Bioblast link«]] | *Liepinsh E, Makarova E, Plakane L, Konrade I, Liepins K, Videja M, Sevostjanovs E, Grinberga S, Makrecka-Kuka M, Dambrova M (2020) Low-intensity exercise stimulates bioenergetics and increases fat oxidation in mitochondria of blood mononuclear cells from sedentary adults. Physiol Rep 8:e14489. — [[Liepinsh 2020 Physiol Rep|»Bioblast link«]] | ||
|} | |} | ||
---- | ---- | ||
Revision as of 08:18, 26 August 2020
2020 Week 35
- Aug-24 Mo
Can NADH be used as an externally added substrate to obtain the NADH electron transfer-pathway state in mitochondrial respiration?
No, in most types of intact mitochondria NADH cannot pass the mitochondrial inner membrane (mtIM). Yeast mitochondria are an exception.
- »Nicotinamide adenine dinucleotide« and »Substrates and metabolites«
- Aug-25 Tu
Low-intensity exercise increases fat oxidation in mitochondria of blood mononuclear cells from sedentary adults
- from the O2k-Network »LV Riga Makrecka-Kuka M«
- Liepinsh E, Makarova E, Plakane L, Konrade I, Liepins K, Videja M, Sevostjanovs E, Grinberga S, Makrecka-Kuka M, Dambrova M (2020) Low-intensity exercise stimulates bioenergetics and increases fat oxidation in mitochondria of blood mononuclear cells from sedentary adults. Physiol Rep 8:e14489. — »Bioblast link«
- Aug-26 We
Non-mitochondrial respiration or residual oxygen consumption (Rox)? — Concept-driven constructive terminology frames our perception. After inhibition of electron-transfer (ET) pathways or in the absence of fuel substrates, both mitochondrial non-ET reactions and non-mitochondrial reactions contribute to residual oxygen consumption in mt-preparations and living cells.
- »BEC: Mitochondrial physiology« — Table 1: Rox induced by non-ET pathway oxidation reactions
- Aug-27 Th
- Aug-28 Fr
