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Difference between revisions of "Template:Current week ecosystem agenda"

From Bioblast
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* '''Aug-24 Mo'''  
* '''Aug-24 Mo'''  
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| width="110" align="center" | [[Image:O2k-Support.jpg|60px|link=http://wiki.oroboros.at/index.php/O2k-Open_Support |O2k-Open Support]][[File:Red-loss of data.png|25px|Warning: loss of data]]
| width="110" align="center" | [[File:MitoPedia.jpg|left|80px|MitoPedia|link=MitoPedia]]
|'''Can NADH be used as an externally added substrate to obtain the [[NADH electron transfer-pathway state]] in [[Mitochondrial_respiration |mitochondrial respiration]]? '''<br>
|'''Can NADH be used as an externally added substrate to obtain the [[NADH electron transfer-pathway state]] in [[Mitochondrial_respiration |mitochondrial respiration]]? '''<br>
No, in most types of intact mitochondria NADH cannot pass the [[mitochondrial inner membrane]] (mtIM). Yeast mitochondria are an exception. <br>
No, in most types of intact mitochondria NADH cannot pass the [[mitochondrial inner membrane]] (mtIM). Yeast mitochondria are an exception. <br>

Revision as of 08:20, 26 August 2020

2020 Week 35
  • Aug-24 Mo
MitoPedia
Can NADH be used as an externally added substrate to obtain the NADH electron transfer-pathway state in mitochondrial respiration?

No, in most types of intact mitochondria NADH cannot pass the mitochondrial inner membrane (mtIM). Yeast mitochondria are an exception.
- »Nicotinamide adenine dinucleotide« and »Substrates and metabolites« 


  • Aug-25 Tu
O2k-Publications in the MiPMap Low-intensity exercise increases fat oxidation in mitochondria of blood mononuclear cells from sedentary adults
from the O2k-Network »LV Riga Makrecka-Kuka M«
  • Liepinsh E, Makarova E, Plakane L, Konrade I, Liepins K, Videja M, Sevostjanovs E, Grinberga S, Makrecka-Kuka M, Dambrova M (2020) Low-intensity exercise stimulates bioenergetics and increases fat oxidation in mitochondria of blood mononuclear cells from sedentary adults. Physiol Rep 8:e14489. — »Bioblast link«

  • Aug-26 We
MitoPedia
Non-mitochondrial respiration or residual oxygen consumption (Rox)? — Concept-driven constructive terminology frames our perception.

After inhibition of electron-transfer (ET) pathways or in the absence of fuel substrates, both mitochondrial non-ET reactions and non-mitochondrial reactions contribute to residual oxygen consumption in mt-preparations and living cells.
- »BEC: Mitochondrial physiology« — Table 1: Rox induced by non-ET pathway oxidation reactions


  • Aug-27 Th

  • Aug-28 Fr